What The Heck is Hyperopia?

One of the most difficult things I have to explain to patients and parents of patients is hyperopia or near sightedness. I must thank my good friend Paul Gooch O.D. for this description of this condition and the difficulties it presents for both patient and practitioner.

Hyperopia (Farsightedness) Explained
After examining a two-year old nephew this week, I attempted to explain what it means to be farsighted to his worried mother. I've been in practice for nearly 12 years and I still struggle to explain hyperopia in lay terms. Its not that Hyperopia is complicated, but it is not as intuitively tidy as Myopia, or nearsightedness (where near things are clear and far things are blurry). Patients often exclaim, "Ah, Farsighted... so things far away are clear and things up close are blurry?" Well, sort of. Therein lies the problem with understanding Hyperopia. We collectively miss-apply the logic that so nicely fits nearsightedness, to farsightedness. And people often confuse presbyopia, or bifocal vision with farsightedness. Even a quick google search on the subject reveals very little for the lay person that isn't slightly off colored just enough to leave an element of confusion. This post is for you, Sis, so you'll grasp how your young son sees; and for me, as I work to conjure a solid explanation of Hyperopia.

The Normal Eye
First, to understand Hyperopia, you must understand how the normal eye functions. When viewing things in the distance (20 feet and beyond are optically equal), the normal eye is relaxed and focused. The light rays entering the eye are parallel, and the relaxed eye converges those parallel rays, bringing them into focus on the retina. When the eye shifts its gaze to anything inside 20 feet, the rays of light begin to diverge, or open up, which pushes the focal point to somewhere behind the normal relaxed eye. The eye then has to accommodate, or shift focus, to keep the near object in focus. This happens when an internal muscle called the cilliary body activates and changes the shape of the crystalline lens that sits behind your pupil. This accommodation [Photo]pulls the long-focused rays back into focus on the retina. And it all happens faster, and more fluidly than you can perceive, without conscious effort on your part. The normal eye uses about 2.50 diopters of accommodative power to see things at the normal reading distance (approx 16 inches).



Hyperopia(Farsightedness)
Hyperopia is the condition where the natural relaxed eye focuses the normal parallel rays from the distance behind the eye. How the farsighted person sees is based on the complex interaction between the amount of farsightedness, the natural strength of the accommodative mechanism, the person's age, and the visual system's alignment mechanisms which keep the eyes on target (they happen to be tied to the focusing system). A young person with small to moderate amounts of Hyperopia can use their close focusing power to pull the distance into focus on the retina and see clearly. Near objects take an additional 2.50 diopters in focus power, and if they have the ability to do it, they can see close as well. Here are some examples of a small and a large amount of Hyperopia:A person with +1.00 diopter Hyperopia needs 1.00 diopter of their close-focus power to see in the distance. Add another 2.50 of accommodation for close vision and they end up needing a total of 3.50 in close focusing power to see clearly at the reading distance.A person with +7.50 diopters of Hyperopia needs 7.50 diopters of help to correct the distance (usually they can't contribute that much by themselves unless they are very young) and they need another 2.50 for near for a total of 10.00 diopters of focusing power.Since the eye's close focusing power was not designed to remain constantly activated, just like your arms weren't designed to hold a bucket of water outstretched for long periods of time, uncorrected Hyperopes can get extra fatigued with the full time, extra-duty use of their close focusing power. Depending on the factors mentioned above, Hyperopes experience a range of symptoms from mild, imperceptible strain, to extreme fatigue, double vision, and completely blurry distance and near vision.
Correction
Hyperopia can be corrected with glasses, contacts, or surgery, using PLUS dioptric power. Unlike Myopic (nearsighted) corrections which are very exact and simple to derive, Hyperopic corrections depend on the practitioner's ability to measure the amount of Hyperopia and then integrate all the other complex factors involved like age, accommodative strength, eye alignment, and lifestyle needs. Generally, correcting Hyperopia relaxes the farsighted eye for focusing in the distance the way it was designed, so the eye can use the normal amount of close focusing power for reading.Because Hyperopic eyes can contribute accommodation to the vision-equation, finding the amount of correction that makes his/her eyes comfortable can be a challenge, and that equation changes over time as the aging eye loses its natural accommodative ability. The older you get, the easier it becomes to find the right amount of correction because your accommodation interferes less. Sometimes younger eyes won't relax and accept plus powered lenses. Large amounts of Hyperopia are easier to correct because the visual benefit is so dramatic. Small to moderate amounts are tougher because the benefit is the delayed gratification of reduced fatigue--which benefit often comes at the end of the day, the end of the week, the end of the school year, etc. For the younger Hyperope, we don't always correct the full amount, but instead, the amount that makes the eyes most comfortable--especially for close work.

Children and Hyperopia
Hyperopia is the most missed eye condition at school and pediatrician screenings because small to moderate amounts don't usually blur the child's vision. Children's symptoms increase with age and amount of Hyperopia, but they often don't know that the discomfort they feel is abnormal and they get numb to it. Often, it plays out as near-task avoidance. If reading is uncomfortable or tiresome after extended periods, the child usually develops avoidance patterns, which can turn into habits, attitudes, labels, and a self perception that can often be difficult to correct as the child gets older. As a parent, you can watch for these avoidance patterns, afternoon headaches, or strange visual behavior like squinting, sideways looking, or looking closer than normal at books, TV, etc.Some eye doctors use a blanket approach to correcting a child's hyperopia--relying strictly on numbers. It is very important to find a practitioner who is committed to using the more sophisticated approach of finding the correction that makes the farsighted child comfortable for near tasks (whether that amount is none, or something different from their full correction). If correction is needed, sometimes single vision lenses suffice, or sometimes it is more convenient to put it in the form of bifocals. These are all complexities that take time to sort through and you will do well to expect your doctor to explain and recommend what is best for your child.Having your child examined before the age of two is the best way to make certain that problems aren't missed which can lead to poor school performance and delayed visual development. You did a wonderful service for your son by bringing him in early, Sis.

I Tried To Save My Glasses.

As a child I think I looked some what like Ralphie and I had about the same luck with glasses as him. Tackle football and glasses just never got along all that well. I know many of you parents can relate to mine as I would come home with pair after pair of destroyed, or worse yet, lost glasses. If you are tired of this as my parents were, now you have another option for your active pre-teen or teenager. Paragon Corneal Refractive Therapy, also known as CRT.

Paragon CRT is a specifically designed therapeutic contact lens worn during sleeping hours to gently reshape the cornea without permanently altering its physiology- which is important, as the eyes of children and teens are still maturing. Your child simply wears the lenses at night and takes them out in the morning. The result is clear vision all day long with no more glasses to lose or break. The procedure has been proven safe and has been approved by the FDA for patients of all ages. I have personally have had great results on children as young as eight. It is a paticularly great option for young atheletes.

Another area where CRT can be of great use is in children who seem to be getting more and more near sighted rapidly. CRT is thought to slow or stop this refractive progression. We have used it successfully in our practice in just this way.

CRT is a great option for adults who are not ready for laser vision correction but would still like to have day time freedom from glasses and contact lenses or are unable to have laser vision correction for a variety of reasons. For more information please give us a call or stop by and see us at Advanced Family Eyecare or visit the link above.

Dr. David I Jones O.D.

Want Longer Lashes? Here you Go.

Early in my career as an Eye Doctor I began to have an experience that would repeat itself many times over the past ten years. I would break the bad news to a woman that I think she has glaucoma, a disease which can cause blindness but is typically successfully treated with eye drops. Xalatan, Travatan and Lumigan are three very similar drops we use to lower eye pressure and decrease the risk of vision loss for patients with glaucoma. I would put her one of these drops and schedule her to come back for regular visits to make sure that every thing was going well and no vision loss was occurring. At those return visits many of these woman would say, Dr. Jones, this is the best thing that has ever happened to me. You see, one of the side effects of the drops is that eye lashes get longer, thicker and darker while using it, some times dramatically so. They now had the lashes they had dreamed of their whole life without using mascara. My wife has even asked jokingly if she could have some.

Well now she can. Allergan, the maker of Lumigan, received FDA approval on December 28, 2008 for a new drop, Lattise with the same active ingredient as Lumigan. Studies have shown that over a 16 weeks course of using the drop lashes have become significantly longer, thicker and darker. The New York Times recently posted this interesting article on Lattise, its cost and potential side effects.

I just spoke with our local Allergan representative and he told me that they will be launching Lattise in March and we should have samples near that time so if you are interested give us a call.

Blind Google Engineer

Enjoy this link to the New York Times article about a Google Engineer who has been blind from Glaucoma from the age of 14. I marvel how people without vision adapt to our very visual world. This is a great example of this adaptation,with practical application.

Regards;

Dr. Gooch

Diabetes and the Ketogenic Diet

News you can use, Click here.

Researchers report (at MedpageToday) the finding that a Ketogenic diet, (one high in fat, and low in Carbohydrates) is more effective in controlling blood glucose than the earlier low-glycemic diet espoused by the Atkins Diabetes Revolution.

The study had some flaws, but the findings are worth a look and raise interesting questions that contribute to our overall understanding.

As eye physicians, we are keenly interested in blood sugar control for diabetic patients. Strict blood sugar control delays or prevents the devastating retinopathy that goes with diabetes. As always, we recommend you take this information and apply it carefully, consulting your internist with all diet and excersize decisions you make that can affect your blood sugar.

Glaucoma explained

According to the World Health Organization, in a 2002 report, Glaucoma is the 2nd leading cause of blindness after Cataracts worldwide. In the United States, Glaucoma is the 2nd leading cause of blindness after Macular Degeneration, closely followed by Diabetic Retinopathy.


What does the average person need to know about Glaucoma? Lets talk about what we do and do not know about this sneaky disease that robs a person's vision by slowly and silently killing the OPTIC NERVE which sends vision signals from the eye to the brain.


First, what we know:

Science and clinical experience have provided us with an excellent ability to treat glaucoma and significantly reduce your risk of blindness from the disease if detected in time. We treat glaucoma with eye drops that lower the internal pressure of the eye. The better we control eye pressure, the more we protect the dying nerve and preserve a person's vision. We also have learned that the earlier we intervene with glaucoma, the easier it is to control or stop ongoing damage to the nerve. Glaucoma seems to have inertia. Advanced cases are reportedly more difficult to control--similar to putting the breaks on a freight train. That perception may be due to a reduced margin for error when glaucoma is at its end stage when there is very little nerve left to save. We do have patients we treat who came to us with very minimal nerve left and were dangerously near blindness, who have managed to keep their remaining vision with consistent treatment.

We know that there are several types of glaucoma. The primary types are those described in this post and are the most difficult to understand. There are several secondary types that happen when the pressure spikes due to anatomical problems, genetic defects, or injury.



What we don't know:

In the past, our view of glaucoma was rather simplistic. Too much pressure in the eye pinched the nerve and slowly strangled it to death. The pressure became elevated due to poor drainage or excessive production of the fluid that is constantly being exchanged in the front of the eye. While this certainly explains some glaucoma cases, there are many that don't fit this tidy scenario. For those of us that battle this disease daily, glaucoma is the Sneak-thief of sight. Sometimes it can look like you have glaucoma when you don't. Conversely, it can look like you don't when you do, and is often missed at Eye Exams. You can have high pressure and no glaucoma, or you can have normal pressure and have glaucoma. And if you have low pressure glaucoma, we treat by lowering the pressure (which is proven to help), but we don't know why it does. When you add to that the fact that you have to lose a significant percentage of your nerve (50-75%) before we can measure damage to your vision via Visual Field testing (one of the traditional keystones to diagnosis), you can begin to appreciate why diagnosing it can get so complicated. Theories abound, but why glaucoma actually happens is still a mystery.

So, the moment glaucoma starts in a patient, the majority of doctors will miss it. The moment you go blind from glaucoma, the majority of doctors will admit that you have it. And there is this large chasm between the two. In medicine, we love to be certain of our selves and our diagnoses which I think lends bias toward later diagnosis since glaucoma doesn't get obvious until its later stages.

Risk factors:

Over the past few years, scientific study has helped us define some of the risk factors associated with the Primary glaucomas. In general, they are listed in order of importance:

• High pressure


• large Nerve cupping


• thin corneas


• Age


• Family History


• African, Asian, or Latin race (but all races affected)


• Severe Myopia (near sighted)


• systemic disease--high blood pressure, diabetes


• Sleep Apnea


• History of Migraines

Early diagnosis:

Because glaucoma is an asymptomatic process, and because it doesn't become obvious until its end stages, there is a movement toward early diagnosis. We know that the earlier we diagnose this disease, the more uncertainty we have to live with, and the more we have to rely on the preponderance of risk factors. In medicine, we commonly treat based on risk factors (we lower cholesterol to reduce your chances of Heart Disease and Stroke, for example.) Thinking of glaucoma this way is a paradigm change from the days when we wanted to declare with absolute certainty that "you did or did not have glaucoma" when we finished up your eye exam.

The tools:

Today, one of the most significant tools we have in our toolkit is the LASER. In our office, we use Ocular Coherence Tomography (OCT) by Humphries Ziess to measure the thickness of your Retinal nerves and track that thickness over time. Thinning nerve suggests glaucoma. We also use high resolution retinal photography over time, we measure your corneal thickness, and the gold-standard Visual field testing over time (which really doesn't tell us too much in the early stages of the disease). We combine the data we collect from these tests with what we know about your risk factors and make our decisions about treatment or followup.

The treatment:

With all the uncertainty about the origins of glaucoma and its early detection, we are very certain about treating the disease. Our goal is to lower the pressure in your eyes, and then monitor the health of your nerve as we keep your pressure down over time. Today's meds are effective and easier than ever. In most cases, one drop of medicine in your eyes each night does the trick. If today's first-line meds don't work, we may have to use additional drops, which may mean instilling drops 2 or 3 times a day. If drops don't work, then we use LASERs and surgery to reduce eye pressure. Effective eye-pressure control protects the health of your nerve and preserves your vision.